Pagkukumpuni ng Katatagan ng AC Joint Impormasyon Pahintulot

Bakit inirerekomenda ang operasyong ito

Ang prosedurang ito ay nagtitiyak ng katatagan sa iyong acromioclavicular (AC) joint, ang koneksyon ng iyong collarbone at shoulder blade. Si Dr. Kieran Hirpara, isang upper-limb surgeon sa Mater Private Hospital Rockhampton, ay pinoproseso ito sa aming klinikang pamamagitan ng pag-aayos ng mga ligament na nag-iikot sa mga butong ito. Karaniwang inirerekomenda namin ito para sa malalaking paghihiwalay (Type IV, V, o VI) kung saan ang mga buto ay nasa maling posisyon. Maaari ring isipin ang operasyon para sa mga sugat na Type III kung ang iyong collarbone ay nasa maling posisyon higit sa 2 cm, o kung ikaw ay isang batang aktibong pasyente na may patuloy na sintomas.

Para sa mga maliit na sugat, karaniwan naming pinagsisimulan ang hindi operasyonal na pag-aalaga tulad ng sling at physiotherapy. Ang operasyon ay sumusunod kapag hindi ito nagbigay ng sapat na pag-unlad o para sa mga akutong structural na problema. Layunin ng operasyong ito na ibalik ang katatagan at bawasan ang sakit, upang makabalik ka sa mga araw-araw na gawain at trabaho.

Bago ang operasyon

Kailangan mo ng pagsusuri para sa anestesia at malamang ay mga X-ray upang maiplan ang iyong operasyon. Inirerekomenda namin na mag-fasting mula sa hating-gabi bago ang iyong prosedura. Itigil ang anumang gamot na nagpapapayat ng dugo lamang pagkatapos kumpirmihin ng iyong doktor kung alin ang mga ito. Dalhin ang kumpletong listahan ng iyong kasalukuyang gamot sa ospital. Mag-ayos ng taong magdadala sa iyo pauwi, dahil hindi ka makakapagmaneho agad. Magsuot ng maluwag at komportableng damit na may bukas sa harap. Mas madali itong isuot kasama ang anumang sling o suporta na ibibigay namin pagkatapos ng iyong operasyon. Mangyaring dumating nang tepat upang maipaghanda ka nang ligtas.

Sa araw ng operasyon

Ang operasyong ito ay isinasagawa sa ilalim ng general na anestesia na pinagsama ng regional na nerve block. Ikaw ay ganap na matutulog habang ginagawa ang operasyon, at ang block — isang injeksyon na nagpapabango sa mga nerbiyong nagbibigay-daan sa braso bago ka gumising — ay nagbibigay ng pagpapagaan ng sakit sa unang 12 hanggang 24 oras pagkatapos ng operasyon. Ang anestesiologist ay makikilala ka bago ang operasyon at ipapaliwanag sa iyo ang parehong bahagi.

Dadating ka sa ospital at aayusin ang iyong pagpasok sa ward. Ang aming koponan ay ihahanda ka para sa operasyon. Ang iyong surgeon ay gagamit ng bukas na paraan na may iisang karaniwang incision sa itaas ng operasyon site. Ito ay nagbibigay ng direktang access sa kasu-kasuan para sa pag-stabilize. Pagkatapos, ikaw ay ililipat sa operating room. Pagkatapos ng proseso, ikaw ay magigising sa recovery area. Ang aming mga nurse ay magm-monitor sa iyong kumportable at vital signs habang ang anestesia ay unti-unting nawawala. Mananatili ka sa recovery hanggang sa ikaw ay stable at handa na bumalik sa ward o pumunta sa bahay, depende sa iyong pag-unlad sa recovery.

Ano ang kinabibilangan ng operasyon

Gagawa ang iyong doktor ng isang hiwa na humigit-kumulang 8 hanggang 10 cm ang haba sa harap ng iyong balikat. Ang paraang ito ay nagbibigay ng malinaw na pananaw sa kasukasuan habang pinapanatili ang mababang pinsala sa tisyu. Maingat naming aayusin ang mga ligamentong napunit na nagtutulak sa iyong collarbone. Para sa mga mas mataas na antas ng pinsala, pinapapalitan namin din ang espasyo sa pagitan ng collarbone at ng shoulder blade bone upang maibalik ang katatagan.

Gagamit kami ng maliliit na anchors o buttons upang siguraduhin ang mga bagong aayus na ligamento. Ito ang magpapanatili sa mga buto sa tamang posisyon habang sila ay gumagaling. Hindi kami gumagamit ng tendon grafts para sa mga akutong pinsala na ginawa sa loob ng 3 hanggang 4 linggo mula sa insidente. Ang hiwa ay isasara pagkatapos gamit ang mga tahi o staples. Isang dressing ang ilalagay upang protektahan ang lugar.

Ang bukas na teknika na ito ay nagpapahintulot sa amin na harapin ang itaas na kasukasuan at ang mas malalim na mga istruktura ng katatagan. Ito ay nakaiiwas sa pangangailangan para sa mga metal implant na maaaring kailangang alisin sa hinaharap. Ang layunin ay maibalik ang natural na pagkakahanay at lakas ng iyong balikat.

Pagkatapos ng operasyon

Gising ka sa recovery ward na may dressing at sling para suportahan ang iyong braso. Pamamahalaan namin ang iyong sakit gamit ang mga pamantayang paraan upang manatili kang komportable. Karamihan sa mga pasyente ay nananatili ng isang gabi sa ospital pagkatapos ng operasyong ito, bagama't may ilang makakapunta sa bahay sa parehong araw. Kailangan mong magkaroon ng kasama na mananatili sa iyo sa loob ng unang 24 na oras upang tumulong sa pag-aalaga. Panatilihing tuyo at malinis ang dressing habang ito ay gumagaling. Huwag magmaneho ng hindi bababa sa ANIM NA LINGGO pagkatapos ng anumang operasyon sa balikat, anuman ang braso na na-operahan. Kailangan mong manatili sa sling sa loob ng panahong ito. Kapag inilaya ka ng iyong surgeon, karaniwan sa pagsusuri sa ika-anim na linggo, maaari ka nang magmaneho. Tingnan ang Pagmamaneho pagkatapos ng surgery sa itaas na bahagi ng katawan para sa kumpletong detalye.

Pagbawi

Maaaring maranasan mong may sakit at pamamaga sa paligid ng iyong balikat sa mga araw pagkatapos ng operasyon. Ito ay bahagi ng normal na proseso ng paggaling. Pinamamahalaan namin ang hindi komportableng pakiramdam na ito gamit ang mga iniresetang gamot pang-alis ng sakit at mga ice pack. Ang pagpapanatili ng iyong braso sa suporta ng sling ay tumutulong upang bawasan ang stress sa mga nakakagaling na tisyu. Karamihan sa mga pasyente ay nakakaramdam na unti-unting bumababa ang antas ng sakit habang bumababa ang paunang pamamaga.

Magbabago ang iyong pang-araw-araw na gawain habang pinoprotektahan ang lugar ng operasyon. Inirerekomenda namin na matulog sa kalahating nakahiga na posisyon upang mapanatili ang iyong balikat na nakataas at komportable. Magdudulot ka ng sling upang limitahan ang galaw habang nagsisimula ang iyong katawan na ayusin ang mga ligamento. Pinapayuhan ang mga banayad na galaw ng kamay at pulso upang mapanatili ang daloy ng dugo, ngunit kailangan mong iwasan ang pag-angat, pagtulak, o paghila gamit ang apektadong braso. Gabay ng iyong pisioterapeuta ang iyong mga partikular na ehersisyo upang muling mabigyan ng lakas at saklaw ng galaw ang iyong braso habang umuunlad ang iyong paggaling.

Hindi ka makakapagmaneho ng hindi bababa sa anim na linggo pagkatapos ng operasyon, anuman ang braso na ginamutan. Kailangan mong manatili sa sling habang ipinagbabawal ang pagmamaneho. Maaari kang bumalik sa pagmamaneho kapag pinapayagan ka ng iyong doktor, karaniwan sa pagsusuri sa anim na linggo. Para sa karagdagang detalye, tingnan ang aming gabay tungkol sa Pagmamaneho pagkatapos ng operasyon sa itaas na bahagi ng katawan.

Iba-iba ang pagbawi sa bawat indibidwal. Maaaring magkaiba ang iyong timeline; gabay ng iyong doktor at pisioterapeuta ang iyong pag-unlad base sa tiyak na proseso ng iyong paggaling at mga layunin sa pagganap.

Maaaring mangyari

Karamihan sa mga pasyente ay magagaling, ngunit minsan ay may mga problema na maaaring mangyari. Ang iyong surgeon at ang team ay magbabantay nang maigi upang maagang matukoy ang anumang isyu.

Maaaring mapansin mo ang patuloy na kahirapan sa joint ng balikat. Karaniwang parang matinding sakit o katigasan na hindi ganap na nawawala. Mas karaniwan ito kung ikaw ay bata at sobrang aktibo. Maaari ring makita mo na nagiging mas masakit ang joint sa paglipas ng panahon, katulad ng arthritis na dulot ng pagkasira. Kung patuloy o lumalala ang sakit na ito, pakipag-usap sa susunod mong review.

Minsan, hindi sapat ang relief na dulot ng unang non-surgical na paggamot. Maaaring magkaroon ka ng patuloy na sintomas na nakakaapekto sa iyong pang-araw-araw na buhay. Sa mga kaso na ito, maaaring kailanganin mong isaalang-alang ang operasyon sa huli. Kung pakiramdam mo ay hindi pa rin gumagaling ang iyong kondisyon, makipag-usap sa iyong surgeon kung ang operasyon ba ang angkop para sa iyo.

Kung pipili ka ng operasyon, may mga karagdagang panganib na dapat isaalang-alang. Ang proseso mismo ay maaaring magdulot ng mga komplikasyon na hindi nangyayari kung pahinga lamang. Maaaring mapansin mo na bumalik ang paglipat ng joint sa labas ng tamang posisyon, na kilala bilang redislocation. Maaaring parang biglang pagsabog o pagbabago sa hugis ng balikat ang nararamdaman mo. Maaari ring makarinig ng click o grind kapag gumagalaw ka ng iyong braso.

Minsan ay mabigo ang hardware na ginagamit upang panatilihin ang joint sa tamang posisyon. Maaaring parang dagdag na sakit o kawalan ng katatagan ang nararamdaman mo. Sa ilang kaso, maaaring lumawak ang mga tunnel ng buto sa paligid ng mga turnilyo, na maaaring magdulot ng pagkaluwag ng repair sa paglipas ng panahon. Kung makikita mo ang bagong pamamaga, dagdag na sakit, o pagbabago sa paraan ng paggalaw ng iyong balikat, makipag-ugnayan sa klinika.

Mayroon ding pagkakataon na hindi nananatiling perpektong tuwid ang joint. Maaaring makita mo ang kitang-kita na bulto o maramdaman na hindi pantay ang pag-upo ng balikat. Kung mararanasan mo ang anumang mga senyales na ito, huwag hintayin ang susunod mong nakadadalaang appointment. Tumawag sa amin upang masuri namin ang iyong balikat at desihin ang pinakamainam na susunod na hakbang.

Ang table ng mga komplikasyon sa pahinang ito ay naglalaman ng karaniwang rates kung gusto mo ng mga detalye.

Kailan tawagan ang aming klinika

Tawagan kami kung may lagnat, lumalalang pamumula o paglabas ng likido mula sa sugat, o biglaang matinding sakit. Pumunta sa emergency kung napansin mong may pamamaga sa binti, hirap sa paghinga, pagkawala ng pakiramdam, o hindi mo na makagalaw ang iyong braso. Ang mga senyales na ito ay nangangailangan ng agad na pagsusuri. Nandito kami upang tulungan kang manatiling ligtas habang nagre-recover ka.

Evidence & references

This is the clinical evidence summary written for health professionals. It is technical, and it lists the research this page was built from. You do not need to read it to understand your treatment or to make a decision about it.

Overview

AC joint separation is a common injury with an unknown incidence [1].
Injury results in progressive disruption of the ligamentous support of the AC joint, beginning with the capsular ligaments and progressing to the CC ligaments [1].
The mechanism of injury is usually direct trauma resulting from a fall on the point of the shoulder [1].
Indirect injuries of the AC joint are rare [1].
Higher grade AC joint injuries result in prominence of the distal clavicle [1].
Localized bruising, swelling, and tenderness are present in acute AC joint injuries [1].
The sternoclavicular joint should be evaluated for swelling, deformity, and tenderness in AC joint injuries [1].
Range of motion and rotator cuff strength typically are normal in chronic AC joint injuries but may be limited in acute injuries secondary to pain [1].
The ability to reduce the deformity with manual pressure can help differentiate nonsurgical versus surgical treatment for higher grade AC joint injuries [1].
Horizontal plane translation of the distal clavicle should be assessed manually and compared with the opposite shoulder [1].
A complete neurologic examination of the upper extremity should be performed to rule out brachial plexus injuries in AC joint trauma [1].
Scapular dyskinesis can be seen with AC joint injuries [1].
Plain radiographs for AC joint evaluation include an AP view of the clavicle, a caudal tilt view, and an axillary view [1].
The axillary view is needed to rule out posterior translation of the distal clavicle [1].
In a normal AC joint, the anterior aspect of the clavicle should lie in the same plane as the anterior aspect of the acromion [1].
The normal coracoclavicular distance between the superior aspect of the coracoid and the inferior clavicle should be between 11 to 13 mm [1].
A fracture of the base of the coracoid process should be ruled out in AC joint injuries [1].
A coracoid fracture can result in superior displacement of the clavicle but an intact CC distance, creating a functionally equivalent AC joint separation [1].
A Zanca view is a modified, underpenetrated AP view with a cephalic tilt of 10° to 15° that gives excellent detail of the distal clavicle [1].
Weighted views include bilateral AP views with a weight tied to the wrists in relaxed standing [1].
Weighted views help distinguish between type II and type III separations but rarely are indicated and often are not clinically helpful [1].
Type I AC joint injury is an AC ligament sprain with intact CC ligaments and normal radiographic CC distance [1].
Type II AC joint injury is an AC ligament rupture with sprained but intact CC ligaments and normal radiographic CC distance [1].
Type III AC joint injury involves disruption of the AC and CC ligaments, characterized by increased CC distance and superior displacement of the clavicle of up to 100% of the clavicle width [1].
The deformity in type III AC joint injuries is reducible [1].
A modification of type III injuries has been proposed consisting of type IIIA (horizontally stable) and type IIIB (horizontally unstable) [1].
Type IV AC joint injury involves disruption of the AC and CC ligaments with posterior displacement of the clavicle, increased CC distance, and the distal clavicle herniated into or through the deltotrapezial fascia [1].
The deformity in type IV AC joint injuries is not reducible [1].
Type V AC joint injury involves disruption of the AC and CC ligaments with greater than 100% superior displacement of the clavicle and a markedly increased CC distance [1].
The deformity in type V AC joint injuries is usually not reducible because of herniation through the deltotrapezial fascia [1].
Type VI AC joint injury involves disruption of the AC and CC ligaments with inferior clavicle displacement, where the distal clavicle lies under the acromion or coracoid process [1].
Type VI AC joint injuries are rare [1].
Nonsurgical treatment is recommended for type I and II AC joint injuries [1].
Good functional outcomes can be expected with nonsurgical treatment for type I and II AC joint injuries [1].
Sling immobilization is followed by gradual active ROM exercises and stretching, then strengthening as tolerated for type I and II AC joint injuries [1].
Most patients regain full shoulder function within 4 to 6 weeks after type I or II AC joint injury [1].
Patients with AC joint injuries are at increased risk for painful AC joint arthritis [1].
Between 30% and 50% of young, very active patients will have mild to moderate residual pain at the AC joint [1].
The recommended treatment for type III AC joint injuries is controversial and depends on patient age and activity level [1].
Multiple retrospective comparative studies have shown good clinical results and return to sport with nonsurgical treatment for type III injuries and no advantage from surgery [1].
Criticisms of studies comparing surgical and nonsurgical treatment for type III injuries primarily relate to the use of antiquated surgical techniques in the surgical groups [1].
Meta-analysis and systematic reviews demonstrate similar outcomes for type III separations managed nonsurgically and surgically [1].
Nonsurgical treatment for type III AC joint injuries resulted in quicker recovery and return to work [1].
Surgical treatment for type III AC joint injuries resulted in an increase in complications [1].
One prospective, randomized trial of type III and V injuries showed better results with nonsurgical treatment when clavicle displacement was less than 2 cm [1].
One prospective, randomized trial of type III and V injuries showed better results with surgical management when the clavicle was displaced more than 2 cm [1].
Nonsurgical treatment for type IV, V, and VI AC joint injuries likely results in substantial residual pain and limited function [1].
Outcomes studies for nonsurgical treatment of type IV, V, and VI AC joint injuries are limited [1].
Surgical treatment is recommended for most patients with type IV, V, and VI AC joint injuries [1].
Surgery is indicated for most acute type IV, V, and VI AC joint separations [1].
Surgery can be performed acutely in selected type III separations in younger, physically active patients [1].
Surgery can be performed acutely in selected type III separations in manual laborers [1].
Surgery can be performed acutely in selected type III separations in patients with cosmetic concerns [1].
Surgery can be performed acutely in selected type III separations in patients with chronic injuries who have persistent symptoms [1].
Surgery is often recommended for patients with type IIIB (horizontally unstable) AC joint separations [1].
Healing of the CC ligaments is reliable without the need for graft augmentation if adequate reduction and stabilization are achieved in acute surgery less than 3 to 4 weeks following injury [1].

Anatomy & Pathophysiology

The mechanism of injury for AC joint separation is usually direct trauma from a fall on the point of the shoulder [1].
Indirect injuries to the AC joint are rare [1].
Injury to the AC joint results in progressive disruption of ligamentous support, beginning with capsular ligaments and progressing to coracoclavicular (CC) ligaments [1].
Range of motion and rotator cuff strength are typically normal in chronic AC joint injuries but may be limited in acute injuries secondary to pain [1].
An axillary view is needed to rule out posterior translation of the distal clavicle [1].
In a normal shoulder, the anterior aspect of the clavicle should lie in the same plane as the anterior aspect of the acromion [1].
A fracture of the base of the coracoid process should be ruled out as it can result in superior displacement of the clavicle with an intact CC distance [1].
Weighted views help distinguish between type II and type III separations but are rarely indicated and often not clinically helpful [1].
Type I AC joint injury involves an AC ligament sprain with intact CC ligaments and normal radiographic CC distance [1].
Type II AC joint injury involves AC ligament rupture with sprained but intact CC ligaments and normal radiographic CC distance [1].
Type III AC joint injury involves disruption of both AC and CC ligaments, characterized by increased CC distance and superior displacement of the clavicle of up to 100% of the clavicle width [1].
Type IV AC joint injury involves disruption of AC and CC ligaments with posterior displacement of the clavicle [1].
In type IV injuries, the distal clavicle is herniated into or through the deltotrapezial fascia and the deformity is not reducible [1].
Type V AC joint injury involves disruption of AC and CC ligaments with greater than 100% superior displacement of the clavicle [1].
In type V injuries, the deformity is usually not reducible because of herniation through the deltotrapezial fascia [1].
Type VI AC joint injury involves disruption of AC and CC ligaments with inferior clavicle displacement [1].
In type VI injuries, the distal clavicle lies under the acromion or coracoid process [1].
Type I injury involves AC ligament sprain without injury to CC ligaments, no AC joint widening, and no clavicular displacement [2].
Type II injuries consist of complete rupture of the AC ligament, CC ligament sprain, widening of the AC joint, and an increase in CC distance by less than 25% compared with the contralateral shoulder [2].
In type III injuries, the AC and CC ligaments are disrupted, the AC joint is widened, and the CC distance is increased 25% to 100% compared with the contralateral shoulder [2].
A type IV AC joint separation is diagnosed when the distal clavicle is displaced posteriorly into the trapezius muscle [2].
Type V injury involves disruption of the deltotrapezial fascia with CC distance increased by more than 100% compared with the contralateral shoulder, and tenting of the overlying skin can result [2].
Type VI injury involves inferior displacement of the clavicle into the subcoracoid space [2].
In type I injuries, AC ligaments are sprained, CC ligaments are intact, and the deltotrapezial fascia is intact [2].
In type II injuries, AC ligaments are disrupted, CC ligaments are sprained, and the deltotrapezial fascia is intact [2].
In type III injuries, AC ligaments, CC ligaments, and the deltotrapezial fascia are all disrupted [2].
In type IV injuries, AC ligaments, CC ligaments, and the deltotrapezial fascia are all disrupted [2].
In type V injuries, AC ligaments, CC ligaments, and the deltotrapezial fascia are all disrupted [2].
In type VI injuries, AC ligaments are disrupted, CC ligaments are intact, and the deltotrapezial fascia is disrupted [2].
The horizontal plane stability of the clavicle is provided by the AC ligaments, specifically the posterior and superior portions [3].
Osteoarthritis of the AC joint is more common with advanced age following degeneration of the intra-articular disk [3].
Arthritic deterioration of the AC joint starts in early middle age [3].
AC joint osteoarthritis is more common in patients engaged in repetitive overhead or lifting activities [3].
Previous low-grade AC joint separations can result in painful arthritis [3].
The radiographic severity of AC joint arthritis does not always correlate with patient symptoms [3].
Patients with AC joint osteoarthritis report activity-related pain localized to the AC joint, with occasional radiation anteriorly or along the trapezius [3].
Pain with heavy lifting or when sleeping on the affected side is reported in AC joint osteoarthritis [3].
Point tenderness is seen at the AC joint in patients with osteoarthritis [3].
Pain at the AC joint with terminal elevation and cross-body motion is often seen in osteoarthritis [3].
Selective injection of anesthetic into the AC joint can confirm the diagnosis of osteoarthritis [3].
Osteophyte formation, sclerotic reaction, and bone cysts are commonly seen on radiographs of AC joint osteoarthritis [3].
Bone and joint edema on MRI correlate with AC joint pain [3].
Distal clavicle osteolysis is a localized hyperemia of the distal clavicle resulting in inflammation, bone resorption, microfractures, and secondary arthritis of the AC joint [3].
Distal clavicle osteolysis is more common in males [3].
Distal clavicle osteolysis is seen in younger patients [3].
Distal clavicle osteolysis is associated with heavy lifting (weight lifters) or repetitive motions [3].
Injuries to the AC joint constitute 9% of shoulder injuries [6].
AC joint injuries are often caused by direct trauma to the shoulder or a fall on an outstretched hand [6].
Younger, physically active athletes are at increased risk for AC joint injuries [6].
Patients involved in contact and extreme sports, as well as high-risk activities such as skiing and cycling, are at increased risk for AC joint injuries [6].
The extent of injury to the AC and coracoclavicular ligaments determines the severity of AC joint separation [6].
The amount and direction of clavicle displacement determines the severity of AC joint separation [6].

Classification

The mechanism of AC joint injury is usually direct trauma from a fall on the point of the shoulder; indirect injuries are rare [1].
In a normal AC joint, the anterior aspect of the clavicle lies in the same plane as the anterior aspect of the acromion [1].
The normal coracoclavicular distance (between the superior aspect of the coracoid and the inferior clavicle) is between 11 to 13 mm [1].
A fracture of the base of the coracoid process should be ruled out as it can result in superior displacement of the clavicle with an intact CC distance, creating a functionally equivalent AC joint separation [1].
Weighted views help distinguish between type II and type III AC joint separations but are rarely indicated and often not clinically helpful [1].
Type IV AC joint injury involves disruption of the AC and CC ligaments with posterior displacement of the clavicle [1].
Type IV AC joint injury is characterized by increased CC distance and the distal clavicle herniated into or through the deltotrapezial fascia [1].
Type V AC joint injury involves disruption of the AC and CC ligaments with greater than 100% superior displacement of the clavicle [1].
Type V AC joint injury is characterized by a markedly increased CC distance [1].
The deformity in type V AC joint injuries is usually not reducible due to herniation through the deltotrapezial fascia [1].
Type VI AC joint injury involves disruption of the AC and CC ligaments with inferior clavicle displacement [1].
Type VI AC joint injuries are rare and result in the distal clavicle lying under the acromion or coracoid process [1].
In 1963, AC joint injuries were initially classified into types I, II, and III [2].
The AC joint classification was expanded in 1984 to include types IV, V, and VI [2].
The classification relies on comparative radiographs of the contralateral shoulder to determine each type [2].
Type I injury involves AC ligament sprain without injury to the CC ligaments, no AC joint widening, and no clavicular displacement [2].
Type V injury is similar to type III except the CC distance is increased by more than 100% compared with the contralateral shoulder due to disruption of the deltotrapezial fascia [2].
Tenting of the overlying skin can result in type V AC joint injuries [2].
In Type I AC joint separation, AC ligaments are sprained, CC ligaments are intact, deltotrapezial fascia is intact, and radiographic CC distance is normal (1.1–1.3 cm) [2].
In Type II AC joint separation, AC ligaments are disrupted, CC ligaments are sprained, deltotrapezial fascia is intact, radiographic CC distance increases by less than 25%, the AC joint appears widened, and the injury is reducible [2].
In Type III AC joint separation, AC ligaments are disrupted, CC ligaments are disrupted, deltotrapezial fascia is disrupted, radiographic CC distance increases by 25%–100%, the AC joint appears widened, and the injury is reducible [2].
In Type IV AC joint separation, AC ligaments are disrupted, CC ligaments are disrupted, deltotrapezial fascia is disrupted, radiographic CC distance is increased, there is posterior clavicle displacement, and the injury is not reducible [2].
In Type V AC joint separation, AC ligaments are disrupted, CC ligaments are disrupted, deltotrapezial fascia is disrupted, radiographic CC distance increases by 100%–300%, and the injury is not reducible [2].
In Type VI AC joint separation, AC ligaments are disrupted, CC ligaments are intact, deltotrapezial fascia is disrupted, radiographic CC distance is decreased, and the injury is not reducible [2].

Clinical Presentation

The sternoclavicular joint should be evaluated for swelling, deformity, and tenderness in AC joint injury [1].
Horizontal plane translation of the distal clavicle should be assessed manually and compared with the opposite shoulder in AC joint injury [1].
A complete neurologic examination of the upper extremity should be performed to rule out brachial plexus injuries in AC joint injury [1].
Scapular dyskinesis can be seen with AC joint injury [1].
An axillary view is needed to rule out posterior translation of the distal clavicle in AC joint injury [1].
Fracture of the base of the coracoid process should be ruled out in AC joint injury as it can result in superior displacement of the clavicle with an intact CC distance [1].
Type II AC joint injury involves AC ligament rupture and sprained but intact CC ligaments with normal radiographic CC distance [1].
Type III AC joint injury involves disruption of the AC and CC ligaments, increased CC distance, and superior displacement of the clavicle of up to 100% of the clavicle width [1].
The deformity in type III AC joint injury is reducible [1].
In type IV AC joint injury, the distal clavicle is herniated into or through the deltotrapezial fascia [1].
The deformity in type IV AC joint injury is not reducible [1].
The deformity in type V AC joint injury is usually not reducible because of herniation through the deltotrapezial fascia [1].
In type VI AC joint injury, the distal clavicle lies under the acromion or coracoid process [1].
Type I AC joint injury involves an AC ligament sprain without injury to the CC ligaments and no AC joint widening or clavicular displacement [2].
Type II AC joint injury consists of complete rupture of the AC ligament, CC ligament sprain, widening of the AC joint, and an increase in CC distance by less than 25% compared with the contralateral shoulder [2].
Type III AC joint injury involves disruption of the AC and CC ligaments, widened AC joint, and increased CC distance of 25% to 100% compared with the contralateral shoulder [2].
Type IV AC joint separation is diagnosed when the distal clavicle is displaced posteriorly into the trapezius muscle [2].
Type V AC joint injury involves disruption of the deltotrapezial fascia and tenting of the overlying skin [2].
Type VI AC joint injury involves inferior displacement of the clavicle into the subcoracoid space [2].
In type I AC joint injury, AC ligaments are sprained, CC ligaments are intact, and the deltotrapezial fascia is intact [2].
In type II AC joint injury, AC ligaments are disrupted, CC ligaments are sprained, and the deltotrapezial fascia is intact [2].
In type III AC joint injury, AC ligaments, CC ligaments, and the deltotrapezial fascia are all disrupted [2].
In type IV AC joint injury, AC ligaments, CC ligaments, and the deltotrapezial fascia are all disrupted [2].
In type V AC joint injury, AC ligaments, CC ligaments, and the deltotrapezial fascia are all disrupted [2].
In type VI AC joint injury, AC ligaments are disrupted, CC ligaments are intact, and the deltotrapezial fascia is disrupted [2].
Patients with AC joint osteoarthritis report activity-related pain [3].
Pain from AC joint osteoarthritis is localized to the AC joint, with occasional radiation anteriorly or along the trapezius [3].
Horizontal stability should be assessed in patients with AC joint osteoarthritis [3].
Selective injection of anesthetic into the AC joint can confirm the diagnosis of AC joint osteoarthritis [3].
Radiographs for AC joint osteoarthritis include an AP view and/or a Zanca view of the shoulder [3].
Osteophyte formation, sclerotic reaction, and bone cysts are commonly seen in AC joint osteoarthritis on radiographs [3].
Distal clavicle osteolysis is associated with heavy lifting or repetitive motions [3].
Examination findings for distal clavicle osteolysis include localized pain, swelling, and tenderness similar to those seen in symptomatic AC joint arthritis [3].
The normal CC distance on an AP radiograph should be less than 11 to 13 mm [3].

Investigations

The mechanism of injury for AC joint separation is usually direct trauma from a fall on the point of the shoulder, while indirect injuries are rare [1].
The sternoclavicular joint should be evaluated for swelling, deformity, and tenderness during AC joint examination [1].
Scapular motion should be carefully assessed as scapular dyskinesis can be seen with AC joint injuries [1].
On an axillary view, the anterior aspect of the clavicle should lie in the same plane as the anterior aspect of the acromion [1].
Type IV AC joint injury involves disruption of the AC and CC ligaments with posterior displacement of the clavicle, increased CC distance, and a deformity that is not reducible [1].
Type V AC joint injury involves disruption of the AC and CC ligaments with greater than 100% superior displacement of the clavicle, a markedly increased CC distance, and a deformity usually not reducible due to herniation through the deltotrapezial fascia [1].
Type I AC joint injury involves an AC ligament sprain without injury to the CC ligaments, no AC joint widening, and no clavicular displacement [2].
Type III AC joint injury involves disruption of the AC and CC ligaments, widened AC joint, and an increase in CC distance of 25% to 100% compared with the contralateral shoulder [2].
Type V AC joint injury involves a CC distance increased by more than 100% compared with the contralateral shoulder due to disruption of the deltotrapezial fascia, with tenting of the overlying skin possible [2].
Radiographic severity of AC joint arthritis does not always correlate with patient symptoms [3].
Pain with heavy lifting or when sleeping on the affected side is reported in patients with AC joint osteoarthritis [3].
Pain at the AC joint with terminal elevation and cross-body motion is often seen in patients with osteoarthritis [3].
Selective injection of anesthetic into the AC joint can confirm the diagnosis of AC joint pathology [3].
An AP view and/or a Zanca view of the shoulder provides good visualization of the AC joint for osteoarthritis evaluation [3].

Treatment

Nonsurgical treatment is recommended for Rockwood type I and II AC joint separations [1].
Good functional outcomes can be expected with nonsurgical treatment of type I and II injuries [1].
Sling immobilization is followed by gradual active range of motion exercises, stretching, and strengthening as tolerated for type I and II injuries [1].
Most patients regain full shoulder function within 4 to 6 weeks following nonsurgical treatment of type I and II injuries [1].
Patients with type I and II injuries are at increased risk for painful AC joint arthritis [1].
Between 30% and 50% of young, very active patients will have mild to moderate residual pain at the AC joint after type I or II injuries [1].
Retrospective studies report persistent symptoms in up to 40% to 50% of patients at 1, 6, and 10 years after type I or II injuries [4].
The arm sling is typically used for approximately 1 week in type I injuries and for 2 to 3 weeks in type II AC joint separations [4].
Patients should refrain from contact sports or heavy lifting for approximately 2 to 3 months until full, painless shoulder range of motion is restored [4].
No evidence supports early surgical management for type I or type II AC joint separations [4].
Distal clavicle resection, either arthroscopic or open, can provide a solution for patients with persistent AC joint inflammation, osteoarthritis, or distal clavicle osteolysis following failed nonsurgical treatment of type I or II injuries [4].
The recommended treatment for type III injuries is controversial and depends on patient age and activity level [1].
Multiple retrospective comparative studies show good clinical results and return to sport with nonsurgical treatment of type III injuries, with no advantage from surgery [1].
Meta-analyses and systematic reviews demonstrate similar outcomes for type III separations managed nonsurgically and surgically [1].
Nonsurgical treatment of type III injuries results in quicker recovery and return to work compared to surgical treatment [1].
Surgical treatment of type III injuries results in an increase in complications compared to nonsurgical treatment [1].
One prospective randomized trial showed better results with nonsurgical treatment for type III and V injuries when clavicle displacement was less than 2 cm [1].
One prospective randomized trial showed better results with surgical management for type III and V injuries when the clavicle was displaced more than 2 cm [1].
A 2018 systematic review and meta-analysis of 5 RCTs and 14 cohort studies found no difference in functional outcome scores between surgical and nonsurgical management of type III injuries [4].
In the nonsurgical group of type III injuries, patients had a faster return to work and sports but an inferior cosmetic appearance [4].
A recent prospective randomized clinical trial found no statistical differences in validated outcome scores or return to preinjury sporting activity between surgical and nonsurgical management of acute type III and IV separations at 1 year follow-up [4].
In a recent prospective randomized trial, the nonsurgical group had faster recovery than the surgical group for type III and IV separations [4].
Five patients (16%) in the nonsurgical group of a recent trial required surgery for persistent symptoms at a mean of 8.7 months [4].
A 2007 survey of 664 members and residency directors from the American Orthopaedic Society for Sports Medicine found that 86.3% preferred an initial trial of nonsurgical management for uncomplicated type III AC joint injuries [4].
Surgical intervention for type III injuries is possible in the subacute or chronic setting if initial nonsurgical treatment fails [4].
Early surgical repair of type III AC joint injuries with or without augmentation results in better patient satisfaction and clinical outcomes compared with delayed reconstruction [4].
A 2016 systematic review found superior functional outcomes in the early surgical group compared with delayed surgery for complete AC joint dislocations involving mostly type III injuries [4].
Partial dislocations or redislocations were found in 26% of cases in the early treatment group compared with 38.1% of cases in the delayed group [4].
The rates of complication were 12.5% in the early surgical group and 17.7% in the delayed surgical group, though differences did not reach statistical significance [4].
Surgery is indicated for most acute type IV, V, and VI separations [1].
Nonsurgical treatment for type IV, V, and VI injuries likely results in substantial residual pain and limited function [1].
Surgical indications for type III separations include younger, physically active patients, manual laborers, patients with cosmetic concerns, or those with chronic injuries who have persistent symptoms [1].
Surgery is often recommended for patients with type IIIB (horizontally unstable) separations [1].
Acute surgery (less than 3 to 4 weeks following injury) of high-grade AC joint separations can be successfully performed without the use of tendon graft if adequate reduction and stabilization are achieved [3].
Delayed reconstruction of AC joint separations requires biologic augmentation, either ligament transfer or tendon grafting, in addition to CC stabilization [3].
Anatomic AC joint reconstructions are biomechanically superior to nonanatomic techniques, such as the Weaver-Dunn procedure [3].
Surgical indications for distal clavicle excision include persistent pain and failure of nonsurgical treatment in patients with AC joint arthritis [3].
Relative contraindications for distal clavicle excision include a previous low-grade separation with persistent horizontal plane instability [3].
Biomechanical evidence suggests a resection of 5 mm is needed to prevent contact between the clavicle and the acromion in the absence of instability [3].
Care should be taken to preserve the posterior and superior AC ligaments during distal clavicle excision [3].
Pain relief from distal clavicle excision is reliable in more than 90% of patients in the absence of instability [3].
Previous traumatic instability is associated with persistent pain in 30% to 40% of cases following distal clavicle excision [3].
One systematic review showed slightly better results with arthroscopic excision than with open distal clavicle excision [3].
Direct comparison studies have shown similar or better results with arthroscopic excision than with open techniques [3].
Between 5 and 10 mm of the distal clavicle should be resected in open distal clavicle resection (Mumford procedure) [3].
Meticulous repair of the deltotrapezial fascia is important in open distal clavicle resection [3].
Resection of the distal clavicle for AC joint arthritis should be limited to 5 to 10 mm of bone [3].
Distal clavicle excision for the management of painful AC joint arthritis has a higher failure rate in patients with a history of previous low-grade AC joint separations [3].
Postoperative care for AC joint reconstruction includes sling immobilization for 6 weeks to limit gravity forces on the operative construct [5].
Hand, wrist, and elbow range of motion exercises, as well as pendulum exercises, are initiated immediately postoperatively following AC joint reconstruction [5].
Physical therapy is begun at 4 weeks with gentle shoulder passive range of motion exercises following AC joint reconstruction [5].
Unrestricted active motion is begun at 6 weeks following AC joint reconstruction [5].
Strengthening exercises are initiated at 10 to 12 weeks following AC joint reconstruction [5].
Subacute rehabilitation (1 to 6 weeks postoperative) involves gradually increasing shoulder range of motion, gentle passive stretching, and reducing sling use as pain permits [3].
Late recovery rehabilitation (more than 6 weeks) involves full shoulder range of motion, stretching, and initiation of rotator cuff, scapular stabilizer, and deltoid strengthening [3].
Heavy weight lifting and return to full activities are performed as tolerated during late recovery, with activity progression modified according to symptoms [3].
Residual pain or soreness can persist for 3 to 4 months after distal clavicle excision and can be aggravated by heavy lifting [3].
Complications associated with nonsurgical management of AC joint separations include persistent pain, crepitus, deformity, swelling at the AC joint, late arthrosis, and persistent instability [8].
Osteolysis of the distal clavicle has been reported as a complication of nonsurgical management [8].
Subacromial erosion can occur with the use of a hook plate [8].
Implant failure and migration resulting in vascular or neurologic injuries have been reported with surgical management [8].
Kirschner wires and pins are not advised for AC joint stabilization due to complications [8].
Aseptic foreign body reaction and erosion of the coracoid or clavicle have been reported with the use of synthetic suture loops [8].
Intrasubstance failure of synthetic grafts has been reported [8].
Early or late fractures of the clavicle or coracoid process have been reported, especially with surgical techniques involving tunnels through the coracoid and/or clavicle [8].
Painful implants related to the hook plate or CC screw usually require a second procedure for implant removal [8].
Ossification of the CC space has been reported as a complication following surgery [8].
Loss of AC joint reduction, persistent pain, and instability can potentially complicate surgical outcomes [8].
Neurologic injuries are rare but can involve nerve root injuries secondary to traction, direct injury to the suprascapular nerve, or injury to the brachial plexus with techniques passing grafts or suture loops under the coracoid process [8].
Adhesive capsulitis, osteomyelitis of the acromioclavicular joint, and upper extremity deep vein thrombosis have been reported as complications of AC joint surgery [8].

Complications

The mechanism of injury is usually direct trauma resulting from a fall on the point of the shoulder; indirect injuries are rare [1].
Higher grade injuries result in prominence of the distal clavicle [1].
Localized bruising, swelling, and tenderness are present in acute injuries [1].
Range of motion and rotator cuff strength typically are normal in chronic injuries but may be limited in acute injuries secondary to pain [1].
Type III injuries are characterized by disruption of the AC and CC ligaments, increased CC distance, and superior displacement of the clavicle of up to 100% of the clavicle width [1].
Type IV injuries are characterized by disruption of the AC and CC ligaments with posterior displacement of the clavicle, increased CC distance, and the distal clavicle herniated into or through the deltotrapezial fascia [1].
Type V injuries are characterized by disruption of the AC and CC ligaments with greater than 100% displacement of the clavicle superiorly, a markedly increased CC distance, and deformity usually not reducible because of herniation through the deltotrapezial fascia [1].
Type VI injuries are characterized by disruption of the AC and CC ligaments with inferior clavicle displacement, resulting in the distal clavicle lying under the acromion or coracoid process [1].
Patients with type I and II injuries treated nonsurgically are at increased risk for painful AC joint arthritis [1].
Between 30% and 50% of young, very active patients will have mild to moderate residual pain at the AC joint after type I and II injuries [1].
Nonsurgical treatment for type III injuries resulted in quicker recovery and return to work compared to surgical treatment [1].
Surgical treatment for type III injuries resulted in an increase in complications compared to nonsurgical treatment [1].
One prospective, randomized trial showed better results with nonsurgical treatment when clavicle displacement was less than 2 cm and better results with surgical management when the clavicle was displaced more than 2 cm [1].
Patients are at risk for recurrent or persistent shoulder symptoms after type I and II injuries [4].
Injury to the AC joint articular cartilage or articular disk can result in shoulder complaints subsequent to the injury [4].
Retrospective studies have reported persistent symptoms in up to 40% to 50% of patients at 1, 6, and 10 years after type I and II injuries [4].
In one study, 27% of patients underwent surgical intervention at a mean of 26 months after type I and II injuries [4].
Distal clavicle resection can provide a potential solution in patients in whom nonsurgical treatment fails secondary to persistent AC joint inflammation or the development of osteoarthritis or distal clavicle osteolysis [4].
A 2018 systematic review and meta-analysis found no difference between surgical and nonsurgical groups in terms of functional outcome scores for type III injuries [4].
Patients in the nonsurgical group for type III injuries had a faster return to work and sports, although with an inferior cosmetic appearance [4].
A recent prospective randomized clinical trial found no statistical differences in validated outcome scores or rates of return to preinjury sporting activity between surgical and nonsurgical groups for acute type III and IV injuries at 1 year follow-up [4].
In a recent prospective randomized clinical trial, five patients (16%) in the nonsurgical group required surgery for persistent symptoms at a mean of 8.7 months [4].
Early surgical repair of type III AC joint injuries with or without augmentation seems to result in better patient satisfaction and clinical outcomes compared with delayed reconstruction [4].
A 2016 systematic review found superior functional outcomes in the early surgical group compared with delayed surgery for complete AC joint dislocation involving mostly type III injuries [4].
The rates of complication were 12.5% in the early surgical group and 17.7% in the delayed surgical group, although the differences did not reach statistical significance [4].
Surgical management is usually indicated for type IV and V AC joint separations given the high likelihood of persistent shoulder pain, dysfunction, and substantial deformity [4].

References

[1] Aaos Comprehensive Orthopaedic Review 3. Disorders of the Acromioclavicular Joint > II. Traumatic Conditions of the AC Joint.

[2] Orthopaedic Knowledge Update Sports Medicine 6. Disorders of the Acromioclavicular Joint, Sternoclavicular Joint, and Clavicle > AC Joint Injuries > Classification.

[3] Aaos Comprehensive Orthopaedic Review 3. Disorders of the Acromioclavicular Joint > III. Atraumatic and Degenerative Conditions of the AC Joint.

[4] Orthopaedic Knowledge Update Sports Medicine 6. Disorders of the Acromioclavicular Joint, Sternoclavicular Joint, and Clavicle > AC Joint Injuries > Management.

[5] Campbell S Operative Orthopaedics 4 Volume Set. ARTHROSCOPIC REPAIR OF POSTERIOR HUMERAL AVULSION OF THE GLENOHUMERAL LIGAMENT > ARTHROSCOPICALLY ASSISTED AC JOINT RECONSTRUCTION > TECHNIQUE 52.28.

[6] Orthopaedic Knowledge Update Sports Medicine 6. Disorders of the Acromioclavicular Joint, Sternoclavicular Joint, and Clavicle > AC Joint Injuries.

[8] Orthopaedic Knowledge Update Sports Medicine 6. Disorders of the Acromioclavicular Joint, Sternoclavicular Joint, and Clavicle > AC Joint Injuries > Complications.