肩胛上神经减压 资料 知情同意
为何建议进行此手术
Mater Private Hospital Rockhampton 的上肢外科医生 Kieran Hirpara 医生,会根据您的病情,从创伤最小的治疗方案开始。患者通常由其全科医生(GP)转诊至我们的诊所;如果物理治疗师建议您就诊,您仍需获得全科医生的转诊,才有资格享受 Medicare 报销。在您的预约就诊中,我们会采集病史,检查您的肩部,并在必要时安排影像学检查或神经测试。肩胛上神经是一条环绕肩部后方的神经,为肩部部分肌肉提供神经支配。当该神经受到压迫时,您可能会感到肩部后方或侧方出现钝痛、酸痛,并伴有相关肌肉的无力或萎缩。这种情况通常首先采用非手术方法治疗,包括物理治疗、抗炎药物以及活动方式的调整。当上述措施未能带来足够的改善,或当有明确因素压迫神经时,我们会考虑手术。手术的目的是解除对该神经的压迫。其目标是缓解您的疼痛,并改善肩部的力量和功能。我们将与您讨论该手术是否适合您,并共同做出决定。
手术前
您的大部分准备工作在抵达医院之前完成。您需要在手术前七小时停止进食和饮水。我们要求七小时而非六小时,以便在手术室排程提前时能让您提前手术。您的外科医生会告知您哪些常规药物需要停用以及何时停用,因此请携带一份您正在服用的所有药物的书面清单。请安排他人在术后驾车送您回家,因为您将无法自行驾驶。请穿着宽松、舒适且易于脱换的衣物。可能需要进行影像学检查(如X光、磁共振成像或超声),以规划手术并准确确认神经受压的位置。如果您有其他基础疾病,可能需要进行血液检查或与麻醉师进行术前评估。
手术当日
您将抵达医院的手术入院病区,在此办理入院手续并做术前准备。术前您将与麻醉医生会面。本手术在全身麻醉联合区域神经阻滞下进行。麻醉医生将在术前与您会面,并向您详细讲解这两部分麻醉方案。
随后,您将被带入手术室进行手术。手术结束后,您将在复苏区苏醒。在麻醉消退期间,护士将在该区域监测您的状况。待您的生命体征稳定后,根据手术类型及恢复情况,您将被转入病房或当日出院。
手术内容
这是一种微创手术。您的外科医生会在肩部周围做几个小切口,包括后方的一个切口,并通过这些切口使用小型摄像头进行操作。摄像头会将肩关节内部的情况显示在屏幕上,因此无需进行大切口。
肩胛上神经穿过肩部后方骨质的狭窄通道。在某些患者中,该处的神经会受到紧绷的组织带或充满液体的囊袋挤压。外科医生利用摄像头找到神经受压的位置,然后松解该紧绷的组织带或切除囊袋,使神经不再受困。神经本身保持原状。如果您同时存在肩部的其他问题,例如肌腱撕裂,外科医生通常可以通过相同的小切口一并处理这些问题。
切口使用缝合线关闭。伤口上会覆盖敷料,敷料需保留约10天。
术后
大多数患者在此手术后需在医院过夜,但部分患者可能当天即可出院。您将在复苏区醒来,在麻醉消退期间,护士会密切观察您的状况。您的肩部可能会感到疼痛或沉重,护士可为您用药以缓解不适。您的手臂将佩戴简易吊带以提供舒适支撑。吊带在清洗和进行锻炼时需取下。回家后最初的24小时内,应有人陪同您。请保持伤口清洁干燥。敷料通常保留约10天;除非我们告知您,否则请勿提前拆除。我们会在复诊时为您更换或拆除敷料。您从手术当天起即可在室内活动,轻柔的手部和肘部活动有助于促进血液循环。您至少六周内不得驾车;一旦外科医生确认您符合驾车条件(通常在六周复查时),请参阅上肢手术后驾车。
恢复
您的肩部在最初几天会感到酸痛并伴有轻微肿胀。随着神经恢复,这种情况会逐渐缓解。休息、将手臂保持在吊带中以及医生开具的止痛药将有助于减轻不适感。肩部周围的小切口会在敷料更换期间愈合,我们会在复诊时进行检查。
起初,您的手臂会佩戴一个简单的吊带以提供舒适感。清洗和进行锻炼时需取下吊带。您的物理治疗师将指导您进行轻柔的活动,并在肩部条件允许的情况下逐步增强力量。您从手术当天起即可在室内活动,轻柔的手部和肘部活动有助于促进血液循环。您至少六周内不得驾驶;一旦外科医生允许(通常在六周复查时),请参阅上肢手术后的驾驶。
随着肿胀消退和活动能力恢复,日常任务会变得更加容易。您会注意到,随着肩部周围肌肉力量的恢复,您的抓握力和提举能力有所改善。大多数人发现疼痛随时间推移而减轻,肩部感觉更加有力。一旦您能无疼痛地使用手臂进行日常活动,您可以根据肩部的感觉,逐步恢复工作和您喜欢的活动。
每个人的恢复情况各不相同。您的时间表可能有所不同,您的外科医生和物理治疗师将在每个步骤为您提供指导。
可能出现的并发症
大多数患者恢复良好,但偶尔可能会出现一些问题。您的外科医生和医疗团队会密切监测您的状况,以便尽早发现任何问题。
正在处理的神经有时可能在手术过程中受到刺激。如果发生这种情况,您可能会注意到肩部或手臂肌肉出现新的无力,或者出现之前没有的麻木或刺痛区域。如果您注意到这些变化,请在下次复诊时告知您的外科医生。
关节镜(微创)肩关节手术存在感染的小概率风险。请留意肩部的小切口。感染的迹象包括伤口周围疼痛加剧、从伤口向外扩散的红肿、局部发热或有液体渗出。您可能会感到发烧。如果您发现这些迹象,请联系诊所;如果您感觉不适,请前往急诊部门。
手术后,有时可能会有液体在伤口下方或附近积聚。这表现为感觉柔软或压痛的肿胀,触诊时可能感觉较硬。最初几天出现少量肿胀属于正常现象。如果肿胀增大、变得疼痛,或者覆盖其上的皮肤看起来紧绷发亮,请告知诊所。
肩关节手术偶尔可能会影响手臂远端的神经。您可能会注意到肘部、前臂、手部或手指出现刺痛、麻木或无力。这种情况通常会自行缓解,简单的措施如休息和温和活动即可。如果症状未缓解或加重,请在复诊时提出。如果问题无法自行缓解,对远端神经进行松解手术是一个选择。
本页上的并发症表格列出了典型发生率,如果您想了解具体数据,请参阅该表。
何时联系我们
大多数术后问题会在早期出现,我们更希望尽早得知。如果您发现发烧、伤口发红或渗出液体且情况恶化,或肿胀持续加重并伴有疼痛,请致电我们。如果您出现突然的剧烈疼痛、小腿肿胀或疼痛、呼吸困难、新的麻木区域,或无法活动手臂,请立即前往急诊。如果您注意到肩部、手臂或手部出现新的无力或麻木感,请致电诊所,以便我们尽早检查。
关于该疾病的更多阅读
本页主要介绍手术本身。关于该手术所治疗的疾病,包括证据显示手术在何时有效、何时无效,将在肩胛上神经病变页面中作更详细的介绍。
Evidence & references
This is the clinical evidence summary written for health professionals. It is technical, and it lists the research this page was built from. You do not need to read it to understand your treatment or to make a decision about it.
Overview
- Arthroscopic decompression of the suprascapular nerve provides excellent visualization and the ability to address concomitant shoulder pathology [1].
- Surgical decompression in the setting of suprascapular neuropathy leads to satisfactory outcomes as evidenced by patient-reported outcomes and return to sport rate [2].
- Suprascapular neuropathy treated with suprascapular nerve decompression significantly improves patient-reported outcomes and is noninferior to similar procedures without suprascapular nerve decompression [3].
- Arthroscopic suprascapular nerve decompression for suprascapular neuropathy at the suprascapular and/or spinoglenoid notch in the absence of major concomitant glenohumeral pathology results in good functional outcomes with significant improvements from before to after surgery [4].
- Release of the spinoglenoid ligament with resultant suprascapular nerve decompression may result in relief of pain and a return of normal shoulder function [5].
- No recommendations regarding suprascapular nerve release in conjunction with rotator cuff repair can be made at this time, and further research is necessary to better delineate the indications in the future [6].
- Decompression of the suprascapular nerve at the spinoglenoid notch did not lead to a better functional outcome compared to repair alone in patients with posterosuperior massive rotator cuff tears and suprascapular neuropathy [7].
- A novel arthroscopic technique for suprascapular nerve decompression uses the superior border of the scapula as a guide, potentially reducing operative time and tissue removal compared to previous methods [8].
- The suprascapular artery must be identified and protected along with the nerve during decompression to ensure safety [9].
- Outcomes after arthroscopic suprascapular nerve release at the suprascapular notch predictably led to successful pain relief and strength improvement in patients presenting with pain and weakness respectively [10].
- Arthroscopic release of the suprascapular nerve can be performed safely and effectively, with all patients showing improvement in postoperative electromyographic findings and marked improvement in pain relief and function [12].
- Suprascapular nerve release does not seem to be justified as an adjunct to rotator cuff repair if preoperative EMG findings document normal suprascapular nerve function [19].
Anatomy & Pathophysiology
Nerve Course and Entrapment Sites
- The suprascapular nerve courses from the upper trunk of the brachial plexus to its motor insertion on the supraspinatus and, more distally, the infraspinatus [17].
- The superior transverse scapular ligament arises from the medial base of the coracoid overlying the suprascapular notch [35].
- The suprascapular artery runs superior to the superior transverse scapular ligament, while the nerve runs deep to it [35].
- Entrapment of the suprascapular nerve at the superior transverse scapular ligament causes denervation of both the supraspinatus and the infraspinatus [35].
- The spinoglenoid ligament overlies the suprascapular nerve at the spinoglenoid notch [35].
- Entrapment, traction, or compression at the spinoglenoid notch causes denervation of the infraspinatus [35].
- At the suprascapular notch, the nerve has little translational freedom as it angles around a confined space [49].
- The angled pathway and limited mobility at the suprascapular notch predispose the nerve to mechanical stretching [49].
- Mechanical stretching at the suprascapular notch may be exacerbated by extreme positions of scapular depression, retraction, or abduction [49].
Anatomical Variations and Predisposing Factors
- Anatomical variations at the suprascapular notch include abnormally oriented subscapularis muscle fibers [22].
- Anatomical variations at the suprascapular notch include an anterior coracoscapular ligament [22].
- Anatomical variations at the suprascapular notch include a calcified superior transverse scapular ligament [22].
- These anatomical variations are predisposing factors for suprascapular nerve entrapment [22].
Mechanisms of Injury
- Suprascapular neuropathy can occur as a result of traction, direct trauma, or extrinsic compression [49].
- Suprascapular neuropathy can occur as part of a more generalized brachial plexus disorder [49].
- Traction of the suprascapular nerve can occur at the suprascapular or spinoglenoid notch secondary to repetitive microtrauma [49].
- Repetitive microtrauma primarily arises from overhead activities such as tennis, volleyball, and weight lifting [49].
- Repetitive microtrauma may lead to direct injury to the nerve [49].
- Repetitive microtrauma may lead to indirect injury by affecting the vascular supply to the nerve [49].
- Intimal damage to the axillary or suprascapular artery may lead to microemboli that become trapped in the vasa nervorum [49].
- Microemboli trapped in the vasa nervorum lead to ischemic injury to the suprascapular nerve [49].
- Direct trauma or indirect trauma during glenohumeral dislocation may result in suprascapular neuropathy [49].
- Direct trauma or indirect trauma during proximal humerus fracture may result in suprascapular neuropathy [49].
- Direct trauma or indirect trauma during scapular fracture may result in suprascapular neuropathy [49].
- Iatrogenic injury to the suprascapular nerve has been reported as a consequence of distal clavicle resection [49].
- Iatrogenic injury to the suprascapular nerve has been reported as a consequence of positioning during spine surgery [49].
- Iatrogenic injury to the suprascapular nerve has been reported as a consequence of transglenoid arthroscopic anterior shoulder stabilization [49].
- Iatrogenic injury to the suprascapular nerve has been reported as a consequence of shoulder arthrodesis [49].
- Iatrogenic injury to the suprascapular nerve has been reported as a consequence of procedures utilizing the posterior approach to the shoulder, such as posterior shoulder stabilization [49].
- Repetitive microtrauma or a single traumatic event is more likely to cause suprascapular neuropathy at the level of the suprascapular notch [49].
- Compression by a ganglion cyst or tumor is a cause of suprascapular neuropathy, particularly at the level of the spinoglenoid notch [49].
- Cystic lesions arising from a labral or capsular tear can compress the nerve along its course over the scapula [17].
- Nerve traction is theorized to arise from chronic overhead athletics [17].
- Nerve traction is theorized to arise from a retracted rotator cuff tear [17].
Pathological Associations and Outcomes
- The exact association and etiology of suprascapular neuropathy in patients with rotator cuff pathology remain unclear [16].
- Complete fatty infiltration of intact rotator cuffs can be caused by suprascapular neuropathy [11].
- Fatty infiltration resulting from suprascapular neuropathy is not reversible [11].
Classification
- Suprascapular neuropathy can present secondary to various etiologies including entrapment or compression [17].
- Nerve traction is theorized to arise from chronic overhead athletics or due to a retracted rotator cuff tear [17].
- Suprascapular nerve neuropathy occurs when the suprascapular nerve is injured along its course from the upper trunk of the brachial plexus to its motor insertion on the supraspinatus and, more distally, the infraspinatus [17].
- Nerve injury can occur from either compression or traction [17].
Clinical Presentation
Symptoms and Signs
- Patients with suprascapular nerve neuropathy usually complain of a dull, aching pain in the posterior and lateral aspects of the shoulder [18].
- Suprascapular neuropathy is a potential source of shoulder pain and functional limitation [17].
- Entrapment of the suprascapular nerve at the suprascapular notch is a cause of shoulder pathology that should be considered in people presenting with wasting of the supraspinatus or infraspinatus muscles, or both [29].
- When the nerve is entrapped at the suprascapular notch, patients present with weakness and atrophy of both the supraspinatus and infraspinatus [18].
- With entrapment at the spinoglenoid notch, symptoms of weakness and atrophy are isolated to the infraspinatus [18].
- Compression of the nerve at the level of the spinoglenoid notch leads to weakness and atrophy of the infraspinatus [18].
Etiology and Risk Factors
- A variety of potential causes of compression have been described, including anomalous transverse scapular ligaments, ganglion cysts, abnormal bony morphology, direct trauma, and traction injury [18].
- The incidence of compressive neuropathy is quite high in the overhead athletic cohort [17].
- Most patients in the overhead athletic cohort do not show clinically relevant deficiencies in function [17].
Diagnosis
- The diagnosis of suprascapular neuropathy is based on a combination of a detailed history, a comprehensive physical examination, imaging, and electrodiagnostic studies [17].
- A detailed history and physical examination along with appropriate workup are paramount to arrive at a diagnosis of suprascapular nerve entrapment [18].
- Shoulder surgeons should consider electrophysiologic evaluation of patients with clinical or radiographic signs of suprascapular neuropathy [20].
- Shoulder surgeons should be cognizant of the parameters that constitute an abnormal electrodiagnostic study [20].
Investigations
- Shoulder surgeons should consider electrophysiologic evaluation of patients with clinical or radiographic signs of suprascapular neuropathy and be cognizant of the parameters that constitute an abnormal study [20].
- In the absence of a well-defined lesion producing mechanical compression of the suprascapular nerve, suprascapular neuropathy should be treated non-operatively [15].
- Anatomical variations at the suprascapular notch, including abnormally oriented subscapularis muscle fibers, anterior coracoscapular ligament, and calcified superior transverse scapular ligament, are predisposing factors for suprascapular nerve entrapment [22].
- The purpose of imaging of the shoulder is to help establish the diagnosis, determine the severity of the pathoanatomy, assist in surgical planning, and enable the surgeon to illustrate the condition of the shoulder to the patient [24].
- Unless a specific research protocol is in place, the temptation to “overimage” should be resisted, obtaining only the scans or reconstructions that are necessary for the care of the patient [24].
- Standardized plain films are almost always sufficient to garner the information needed for shoulder evaluation [24].
- The first key radiographic view is the anteroposterior (AP) in the plane of the scapula taken so that the x-ray beam passes through the glenohumeral joint [24].
- The second key radiographic view is the axillary view taken with the arm in the functional position of elevation in the plane of the scapula and oriented so that both the spinoglenoid notch and the scapular neck are visible [24].
- At least two X-ray views should be obtained for shoulder imaging: an anteroposterior in the plane of the glenoid and an axillary projection with the arm in abduction to show the relationship of the humeral head to the glenoid [40].
- Magnetic resonance imaging (MRI) is useful to identify osteonecrosis of the humeral head, or a bone tumour [40].
- MRI can also identify labral tears and rotator cuff tears, although the accuracy for these latter two is enhanced by combining the scan with arthrography [40].
- Computed tomography (CT) is helpful for planning fracture surgery and shoulder joint replacement [40].
- Ultrasound is a simple and accurate test for identifying rotator cuff tears and calcific tendinitis [40].
- Ultrasound can also be useful in guiding injections or barbotage (aspirating calcific deposits in the rotator cuff) [40].
- Arthroscopy is useful for diagnosing and treating subacromial impingement, intra-articular lesions, detachment of the glenoid labrum and rotator cuff tears [40].
Treatment
Non-Operative Management
- Initial treatment of isolated suprascapular neuropathy typically consists of physical therapy, nonsteroidal anti-inflammatory drugs, and activity modification [14].
Operative Indications
- Open or arthroscopic operative intervention is warranted when there is extrinsic nerve compression or progressive pain and/or weakness [14].
- Surgical decompression is indicated in cases refractory to conservative management [18].
Surgical Techniques
- The anterior release of the suprascapular nerve is a feasible alternative to the lateral approach [26].
- Open decompression at the spinoglenoid notch permits direct visualization of the suprascapular nerve and allows for a safe, reliable, and thorough decompression [18].
- For open spinoglenoid notch decompression, the patient is placed in the lateral decubitus position with the head elevated 30° and the body over-rotated 10° to 20° past parallel anteriorly [50].
- During open spinoglenoid notch decompression, the deltoid is split in line with its fibers beginning approximately 4 to 5 cm from the posterolateral corner of the acromion [50].
- The spinoglenoid ligament is released sharply from the edge of the scapular spine during open decompression [50].
Safety Considerations
Clinical Outcomes
- Suprascapular neuropathy treated with suprascapular nerve decompression significantly improves patient-reported outcomes and is noninferior to similar procedures without decompression [3].
- Arthroscopic management of suprascapular neuropathy provides patients with significant improvements in pain, strength, and subjective function of the shoulder, and has a low incidence of complications [13].
Concomitant Pathology
Complications
- Arthroscopic management of suprascapular neuropathy has a low incidence of complications [13].
- Ultrasound-assisted intralesional methylene blue injection may facilitate uneventful decompression of symptomatic spinoglenoid notch cysts while preventing iatrogenic suprascapular nerve injury [51].
- A malpositioned superior screw from reverse shoulder arthroplasty can cause suprascapular nerve entrapment [30].
- Baseplate screw penetration in reverse total shoulder arthroplasty poses a risk of iatrogenic suprascapular neuropathy by screw violation [31].
- Failure to treat multiple diagnoses in the same shoulder can lead to missed pathologic features and the necessity for revision operations [21].
Recovery
- Surgical decompression for suprascapular neuropathy leads to satisfactory outcomes as evidenced by patient-reported outcomes and return to sport rate [2].
- Suprascapular neuropathy treated with suprascapular nerve decompression significantly improves patient-reported outcomes [3].
- Suprascapular nerve decompression is noninferior to similar procedures without suprascapular nerve decompression [3].
- Arthroscopic suprascapular nerve decompression at the suprascapular and/or spinoglenoid notch in the absence of major concomitant glenohumeral pathology results in good functional outcomes [4].
- Arthroscopic suprascapular nerve decompression at the suprascapular and/or spinoglenoid notch in the absence of major concomitant glenohumeral pathology results in significant improvements from before to after surgery [4].
- Release of the spinoglenoid ligament with resultant suprascapular nerve decompression may result in relief of pain [5].
- Release of the spinoglenoid ligament with resultant suprascapular nerve decompression may result in a return of normal shoulder function [5].
- Outcomes after arthroscopic suprascapular nerve release at the suprascapular notch predictably led to successful pain relief in patients presenting with pain [10].
- Outcomes after arthroscopic suprascapular nerve release at the suprascapular notch predictably led to strength improvement in patients presenting with weakness [10].
- Patients experienced immediate improvement in pain following treatment for suprascapular neuropathy [11].
- Patients experienced immediate improvement in subjective shoulder value following treatment for suprascapular neuropathy [11].
- Fatty infiltration was not reversible following treatment for suprascapular neuropathy [11].
- All patients showed improvement in postoperative electromyographic findings after arthroscopic release of the suprascapular nerve [12].
- All patients showed marked improvement in pain relief after arthroscopic release of the suprascapular nerve [12].
- All patients showed marked improvement in function after arthroscopic release of the suprascapular nerve [12].
- Full recovery of shoulder function was achieved in a case of suprascapular nerve entrapment caused by a large hematoma of the scapula [25].
Key Evidence
- [L5] Arthroscopic decompression of the suprascapular nerve provides excellent visualization and the ability to address concomitant shoulder pathology. [1] (10.1016/j.jse.2010.01.006)
- [L4] Surgical decompression in the setting of suprascapular neuropathy leads to satisfactory outcomes as evidenced by the patient-reported outcomes and return to sport rate. [2] (10.1016/j.jse.2017.09.025)
- [L4] Suprascapular neuropathy treated with SSND significantly improves patient-reported outcomes and is noninferior to similar procedures without SSND. [3] (10.1016/j.xrrt.2024.05.007)
- [L4] Arthroscopic SSN decompression for suprascapular neuropathy at the suprascapular and/or spinoglenoid notch in the absence of major concomitant glenohumeral pathology results in good functional outcomes with significant improvements from before to after surgery. [4] (10.1016/j.arthro.2020.10.020)
- [L4] Release of the spinoglenoid ligament with resultant suprascapular nerve decompression may result in relief of pain and a return of normal shoulder function. [5] (10.1177/03635465990270062101)
- [L4] No recommendations regarding suprascapular nerve release in conjunction with rotator cuff repair can be made at this time, and further research is necessary to better delineate the indications in the future. [6] (10.1016/j.jse.2011.11.033)
- [L3] Decompression of the suprascapular nerve at the spinoglenoid notch did not lead to a better functional outcome compared to repair alone in patients with posterosuperior massive rotator cuff tears and suprascapular neuropathy. [7] (10.1186/s12891-021-04075-1)
- [L4] The study describes a novel arthroscopic technique for suprascapular nerve decompression that uses the superior border of the scapula as a guide, potentially reducing operative time and tissue removal compared to previous methods. [8] (10.1007/s00167-009-0858-1)
- [L4] The authors emphasize that the suprascapular artery must be identified and protected along with the nerve during decompression to ensure safety. [9] (10.1016/j.jse.2008.08.007)
- [L4] Outcomes after arthroscopic suprascapular nerve release at the suprascapular notch predictably led to successful pain relief and strength improvement in patients presenting with pain and weakness respectively. [10] (10.1177/2325967123s00003)
- [L4] Patients experienced immediate improvement in pain and subjective shoulder value, though fatty infiltration was not reversible. [11] (10.1016/j.arthro.2014.01.010)
- [L4] Arthroscopic release of the suprascapular nerve can be performed safely and effectively, with all patients showing improvement in postoperative electromyographic findings and marked improvement in pain relief and function. [12] (10.1016/j.arthro.2006.10.003)
- [L4] Results indicate that arthroscopic management provides patients with significant improvements in pain, strength, and subjective function of the shoulder, and has a low incidence of complications. [13] (10.1007/s00167-017-4694-4)
- [L5] Initial treatment of isolated suprascapular neuropathy is typically nonoperative, consisting of physical therapy, nonsteroidal anti-inflammatory drugs, and activity modification; however, open or arthroscopic operative intervention is warranted when there is extrinsic nerve compression or progressive pain and/or weakness. [14] (10.2106/jbjs.i.01743)
- [L4] In the absence of a well-defined lesion producing mechanical compression of the suprascapular nerve, suprascapular neuropathy should be treated non-operatively. [15] (10.2106/00004623-199708000-00007)
- [L3] The exact association and etiology of suprascapular neuropathy in patients with rotator cuff pathology remain unclear. [16] (10.1016/j.jse.2013.06.011)
- [L5] [17] (10.5435/jaaos-d-19-00526)
- [L5] [18] (10.1016/j.eats.2024.103051)
- [L2] Suprascapular nerve release does not therefore seem to be justified as an adjunct to RC repair if preoperative EMG findings document normal suprascapular nerve function. [19] (10.1016/j.jse.2020.03.051)
- [L4] Shoulder surgeons should consider electrophysiologic evaluation of patients with clinical or radiographic signs of suprascapular neuropathy and be cognizant of the parameters that constitute an abnormal study. [20] (10.1016/j.jse.2010.10.039)
- [L4] A complete history and physical, careful attention to auxiliary tests, and treatment of multiple diagnoses in the same shoulder avoids missed pathologic features and necessity for revision operations. [21] (10.1097/01.blo.0000063791.32430.59)
- [L4] Anatomical variations at the suprascapular notch, including abnormally oriented subscapularis muscle fibers, anterior coracoscapular ligament, and calcified superior transverse scapular ligament, are predisposing factors for suprascapular nerve entrapment. [22] (10.1007/s00167-003-0378-3)
- [Case_report] Full recovery of shoulder function was achieved. [25] (10.1186/s12891-023-06723-0)
- [L5] The anterior release of the suprascapular nerve is a feasible alternative to the lateral approach. [26] (10.1016/j.eats.2024.103237)
- [L5] Entrapment of the SSN at the suprascapular notch is a cause of shoulder pathology that should be considered in people presenting with wasting of the supraspinatus or infraspinatus muscles, or both. [29] (10.1016/j.jse.2010.12.003)
- [Case_report] This case is the first report of malpositioned superior screw from reverse shoulder arthroplasty causing suprascapular nerve entrapment. [30] (10.1016/j.jse.2009.10.004)
- [L3] [31] (10.1016/j.jse.2021.10.024)
- [L5] [49] (10.5435/00124635-199911000-00002)
- [L4] [50] (10.1016/j.jse.2013.03.009)
- [L4] We believe that this technique may facilitate uneventful decompression of symptomatic spinoglenoid notch cysts while preventing iatrogenic suprascapular nerve injury. [51] (10.1016/j.jse.2021.03.076)
References
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