Education · shoulder

Calcific Tendinitis Info In-depth Evidence

Reviewed by Dr Kieran Hirpara, Specialist Orthopaedic Surgeon Last reviewed

Also on YouTube.

Video transcript

Calcific tendinitis happens when a small deposit of calcium forms inside one of the rotator cuff tendons in the shoulder. While it is forming it can be quietly uncomfortable, but when the body starts to break it down, the pain can become sudden and severe. The shoulder may feel hot and intensely sore, and moving it can be very hard for a while. It often arrives without warning, and without any injury. The deposit usually shows up clearly on an x-ray or an ultrasound. Encouragingly, these deposits often dissolve on their own with time. In the meantime, anti-inflammatory medication and gentle movement help you through the painful spell. Under ultrasound guidance, the deposit can be softened and drawn out through a fine needle, in a procedure called barbotage. A cortisone injection at the same time can settle the surrounding inflammation. For most people, these steps calm the shoulder without an operation. If a stubborn deposit keeps causing trouble, it can be removed with keyhole surgery. It is done with a camera and instruments through small incisions. The deposit is located within the tendon and cleared away, and the tendon is smoothed and repaired if needed. Any inflamed tissue around it is tidied up at the same time. The arm rests in a sling afterwards. Recovery depends on what was needed, and is often quicker than a full tendon repair. After barbotage, the sharp pain usually eases within days, as the inflammation settles. After surgery, the arm is rested in a sling for comfort, and physiotherapy starts gently to restore movement. Most people regain their range over the following months. The relief once the deposit is gone is usually lasting.

Calcific Tendinitis: Causes, Treatment and Recovery

What you're feeling

You may notice pain in the front or side of your shoulder. It often feels deep inside the joint. This pain can flare up suddenly and feel intense. You might also experience a dull ache that lingers after activity.

It is important to know that not all calcium deposits cause pain. Only one third of calcific tendon deposits in the shoulder are painful. Many people have these deposits without any symptoms at all. If you do have pain, it is often linked to wear-and-tear of the tendons around your shoulder blade.

Your pain may worsen when you lift your arm above your head. Simple tasks like reaching for a cup on a high shelf can become difficult. You might feel stiffness when you try to comb your hair or reach behind your back to fasten a coat. Lifting even light objects, such as a kettle or a bag of groceries, may trigger sharp discomfort.

Night pain is common. You might find it hard to lie on the affected side. The pain can keep you awake or wake you up suddenly. You may also feel stiff when you first wake up in the morning. This stiffness often eases as you move around, but activity later in the day can bring it back.

The severity of your symptoms can depend on the size of the deposit. Patients with calcific lesions larger than 1 cm have a 2.8 increased likelihood to undergo operative treatment. This suggests that larger deposits are more likely to cause significant issues that do not resolve with rest alone.

Most cases improve with conservative treatment. Conservative treatment for calcific tendinitis of the shoulder showed clinically significant improvement, with 72% of excellent or good results regardless of the location, radiologic type and size, and initial symptoms of calcific deposits. This means that for most people, non-surgical approaches like rest, medication, or therapy can provide substantial relief.

However, some people experience persistent pain. If your pain remains severe despite conservative care, your surgeon may discuss other options. These can include procedures to remove the calcium deposit or shock wave therapy to break it down. The goal is always to reduce your pain and restore your ability to use your shoulder for daily tasks.

What's actually happening

Calcium deposits often form in the tendons of your shoulder without you ever noticing. These build-ups are quite common in the general population. However, only one third of these deposits cause any pain at all. You might have them and never know.

When they do hurt, it is because the calcium sits inside a tendon. This is a strong rope-like tissue that connects your muscle to your bone. The deposit acts like a hard pebble inside a soft sleeve. It rubs against the surrounding tissue and creates inflammation. This swelling presses on nearby nerves and causes significant discomfort.

The pain often feels sharp or deep in the shoulder. It can make it hard to lift your arm or reach behind your back. In some cases, the body tries to break down the calcium on its own. This natural process can actually increase pain temporarily before things improve.

Your surgeon will look at scans to see where the calcium is. They check if it is near the surface or deep inside the tendon. The size of the deposit matters too. Lesions larger than 1 cm are more likely to need surgery if conservative treatments do not work.

Sometimes, a tear in the rotator cuff happens alongside the calcium. This tear is the main factor that affects how well your shoulder function recovers. It is important to check for this because it changes the treatment plan.

We also look at the space under your collarbone. Some theories suggested that a high bone index here causes more pain. However, evidence shows this is not the case. The space looks similar whether you have calcific tendinitis or other shoulder issues.

Understanding what is happening helps us choose the right path. Most people improve with non-surgical care. This includes rest, anti-inflammatory medicines, or shock wave therapy. If the deposit is large or persistent, we may recommend a small keyhole surgery to remove it. This clears the blockage and lets the tendon heal properly.

What we can do about it

Dr Kieran Hirpara, an upper-limb surgeon at Mater Private Hospital Rockhampton, starts with the least invasive options that suit your condition. Calcific tendon deposits are common, but only one third cause pain. If you are in that group, we begin with self-management and physiotherapy. We aim to reduce inflammation and restore movement. You will likely try oral anti-inflammatory medication and specific exercises. This approach is the mainstay of treatment for most patients. We also consider ultrasound-guided needling (barbotage) or extracorporeal shock wave therapy (ESWT) as first-line treatments. ESWT uses sound waves to break down deposits. It is safe, well-tolerated, and very effective for chronic cases. Radial ESWT can improve shoulder function by 43% and reduce pain by 14.28% compared to standard physiotherapy alone. We give these conservative measures time to work before considering further steps.

If pain persists, we move to medical management. Corticosteroid injections can provide significant short-term relief by calming the inflamed tissue. These injections are often combined with needling to help flush out the calcium deposits. Needle aspiration of calcific deposits is safe and effective for relieving pressure in the tendon. While some clinics offer hyaluronic acid or PRP, our evidence base highlights corticosteroids and needling as the primary medical interventions for this condition. We monitor your response closely. If symptoms are protracted, they can impact your quality of life and time off work, so we adjust the plan to get you back to normal activities as soon as safely possible.

Surgery is considered only when conservative care has not given enough improvement. We perform arthroscopic debridement, which is a keyhole procedure to remove the calcium deposits and smooth the tendon. This is safe and effective, even if the calcium has spread into the bone. Before any procedure, we conduct a full assessment including imaging to confirm the diagnosis and rule out other issues like rotator cuff tears, which can sometimes be hard to spot on scans when calcific tendinitis is present. Patients are generally referred to our clinic by their GP; if a physiotherapist has suggested you see us, you will still need a referral from your GP in order to be eligible for the Medicare rebate. A clinic assessment (history, examination, and imaging where needed) establishes the diagnosis. For degenerative or long-standing problems we usually try non-operative care — activity change, physiotherapy or hand therapy, splinting, and injections — and consider surgery when that has not given enough improvement. For structural or acute problems, surgery may be recommended straight away, without a preceding non-operative trial.

What to expect

Calcific deposits in your shoulder are common, but they do not always cause pain. In the general population, only one third of these deposits are painful. If you are experiencing symptoms, the first step is usually conservative management. This approach involves non-surgical treatments and time. For many people, this leads to significant improvement. Research shows that 72% of patients achieve excellent or good results with conservative care, regardless of where the deposit is located or how large it is.

Your recovery depends on the stage of the condition. The calcium deposits go through a natural cycle. Sometimes they break down and are absorbed by your body on their own. This resorption process is often linked to symptom duration and deposit size. For example, extracorporeal shock wave therapy (ESWT) works best if your symptoms have lasted 10 months or less and the deposit is 10.82 mm or smaller. If you have had symptoms for longer or have larger deposits, other treatments may be more suitable.

If conservative methods do not provide enough relief, we may discuss procedural options. These include needle aspiration or arthroscopic surgery to remove the calcium. Needle aspiration can reduce pain noticeably within two months. Arthroscopic removal focuses on clearing the deposit completely. The key to a good outcome is whether any calcium remains in the tendon. If the deposit is fully removed, your functional recovery tends to be strong.

It is important to know that calcific tendinitis can coexist with rotator cuff tears. These tears are more common in people with calcific tendinitis than previously thought. We will carefully assess your shoulder to check for any associated tears. This ensures we address all sources of your pain.

Overall, the outlook is positive. Most people improve with appropriate treatment. Some may need a combination of therapies over several weeks or months. We will guide you through the options that fit your specific situation, aiming for a return to your normal activities with minimal discomfort.

When to see someone

Calcific deposits in the shoulder are common, but only one third cause pain. You may have these deposits even if you feel no discomfort. However, if you experience persistent pain, weakness, or instability, it is time to seek help. Sudden worsening of symptoms or pain that interferes with your sleep or work also warrants attention. Be aware that locking or giving way can signal a rotator cuff tear, which sometimes occurs alongside calcific tendinitis. Early assessment helps your surgeon determine if the deposits are the true source of your subacromial pain syndrome. Do not ignore symptoms that limit your daily activities.

Advanced reading: the deeper science (optional)

This section goes further than you need for your own treatment decisions. Calcific tendinitis is worth the extra reading because it behaves unlike most shoulder conditions: the deposit is common in people with no pain at all, the body usually dissolves it without help, and the treatment that removes the calcium is not the one that relieves the pain fastest.

The deposit is a common finding, not automatically a diagnosis

A calcium deposit reported on your scan is easy to read as the cause of the problem, because it is visible and the pain is real. The prevalence data complicate that. Among 1,219 adults, deposits were present in 7.8% of people without symptoms and 42.5% of those with subacromial pain syndrome [1]. In a separate series of 302 shoulders, deposits were frequent in the general population and only about one third were painful [2].

So the deposit raises the probability that it is the source, substantially — but roughly one in thirteen painless shoulders has one too. What appears to shift a deposit from silent to symptomatic is partly size and position: the highest likelihood of genuinely symptomatic disease was in women aged 30 to 60 with subacromial pain and a deposit longer than 1.5 cm [1], and pain correlated with location in supraspinatus and with involvement of more than one tendon [2].

It usually dissolves, and that shapes everything

The natural course is towards resorption, which is why so many treatments appear to work. The clearest illustration comes from a randomised trial in which every patient had needling and lavage and was then randomised to a steroid or saline injection: at twelve months the calcification had resorbed in 83% of the saline group and 74% of the steroid group [3].

Read that carefully, because it contains the counter-intuitive result. The steroid improved pain for six weeks and function for three months — and had no significant effect on whether the calcium disappeared [3]. Pain relief and calcium clearance are separate processes. A treatment can deliver one without the other, and the visible thing on the scan is not the thing driving the symptoms week to week.

Which is why the non-operative options perform alike

If the deposit largely resolves regardless, the treatments are competing to make the interval tolerable rather than to cure. That is what the comparisons show. Across 257 patients, physical therapy, corticosteroid injection and ultrasound-guided barbotage produced similar rates of avoiding surgery [4]. In 239 patients the same three approaches were largely successful, with physiotherapy alone carrying the highest failure rate [5].

Among the procedural options, high-energy shockwave therapy is the most thoroughly studied minimally invasive treatment and has been shown safe and effective in the short to medium term, while ultrasound-guided needling has not been shown superior to an ultrasound-guided subacromial injection [6]. Pooling 1,258 patients, shockwave, needling and arthroscopy all produced good clinical outcomes [7], and barbotage across 908 patients was safe with a high success rate but has never been compared head-to-head against the other major options [8].

What surgery adds, stated precisely

Surgery is not without an advantage, and the size of it is worth quoting rather than characterising. Pooling 2,352 patients from randomised trials, surgical treatment produced larger improvement in functional scores and comparable pain reduction to non-operative treatment, particularly ultrasound-guided needling — with both routes achieving clinically significant improvement [9]. Among surgical techniques themselves there was no significant difference, and removing the deposit alone performed similarly to removing it plus a subacromial decompression [10].

The function-versus-pain split is the useful detail. If pain is the dominant complaint, the evidence does not clearly favour an operation. If stiffness and loss of function dominate and have persisted, it favours one more.

The association most often missed

Calcific tendinitis is not evenly distributed. In 102 patients, those with an associated endocrine disorder — thyroid disease and diabetes principally — developed symptoms at a younger age, had a significantly more protracted course, and more frequently required surgery [11].

This is worth raising with your GP if your course has been unusually long or began early, not because treating the endocrine condition resolves the shoulder, but because it changes what a realistic timeline looks like. Being told a condition is self-limiting is difficult to reconcile with two years of pain; the endocrine association is one explanation for why the usual reassurance does not fit every case.


References for the advanced reading
  1. Louwerens JK, Sierevelt IN, van Hove RP, van den Bekerom MP, van Noort A. Prevalence of calcific deposits within the rotator cuff tendons in adults with and without subacromial pain syndrome: clinical and radiologic analysis of 1219 patients. J Shoulder Elbow Surg. 2015;24(10):1588-93.
  2. Sansone V, Consonni O, Maiorano E, Meroni R, Goddi A. Calcific tendinopathy of the rotator cuff: the correlation between pain and imaging features in symptomatic and asymptomatic female shoulders. Skeletal Radiol. 2015;45(1):49-55.
  3. Darrieutort-Laffite C, Varin S, Coiffier G, Albert J, Planche L, Maugars Y, et al. Are corticosteroid injections needed after needling and lavage of calcific tendinitis? Randomised, double-blind, non-inferiority trial. Ann Rheum Dis. 2019;78(6):837-43.
  4. Gilbert R, Dadoo S, Lin R, Bhardwaj N, McMahon S, Steuer F, et al. Comparison of physical therapy, corticosteroid injections, and ultrasound-guided barbotage for nonoperative management of calcific tendinitis. Orthop J Sports Med. 2026;14(4).
  5. Drummond M, Ayinon C, Lin A, Dunn R. Relative efficacy of three nonsurgical treatments for calcific tendinitis: physical therapy vs steroid injection vs barbotage. Orthop J Sports Med. 2021;9(7_suppl4).
  6. Louwerens JK, Sierevelt IN, van Noort A, van den Bekerom MP. Evidence for minimally invasive therapies in the management of chronic calcific tendinopathy of the rotator cuff: a systematic review and meta-analysis. J Shoulder Elbow Surg. 2014;23(8):1240-9.
  7. Louwerens JK, Veltman ES, van Noort A, van den Bekerom MP. The effectiveness of high-energy extracorporeal shockwave therapy versus ultrasound-guided needling versus arthroscopic surgery in the management of chronic calcific rotator cuff tendinopathy: a systematic review. Arthroscopy. 2015;32(1):165-75.
  8. Gatt DL, Charalambous CP. Ultrasound-guided barbotage for calcific tendonitis of the shoulder: a systematic review including 908 patients. Arthroscopy. 2014;30(9):1166-72.
  9. Angileri HS, Gohal C, Comeau-Gauthier M, Owen MM, Shanmugaraj A, Terry MA, et al. Chronic calcific tendonitis of the rotator cuff: a systematic review and meta-analysis of randomized controlled trials comparing operative and nonoperative interventions. J Shoulder Elbow Surg. 2023;32(8):1746-60.
  10. Anam E, Zahran S, Roy A, Daneshvar P, Bicknell RT, Janssen I. Surgical approaches of shoulder calcific tendonitis: a systematic review and meta-analysis. JSES Rev Rep Tech. 2024;4(3):353-8.
  11. Harvie P, Pollard TC, Carr AJ. Calcific tendinitis: natural history and association with endocrine disorders. J Shoulder Elbow Surg. 2007;16(2):169-73.
Evidence & references

This is the clinical evidence summary written for health professionals. It is technical, and it lists the research this page was built from. You do not need to read it to understand your treatment or to make a decision about it.

Overview

  • Calcific tendon deposits of the shoulder are a frequent occurrence in the general population [1].
  • Only one third of calcific tendon deposits are painful [1].
  • The primary choice of treatment for calcific tendinitis is conservative, especially in patients with acute calcific tendinitis [7].
  • Conservative treatment for calcific tendinitis of the shoulder showed clinically significant improvement, with 72% of excellent or good results regardless of the location, radiologic type and size, and initial symptoms of calcific deposits [3].
  • Patients with calcific lesions >1 cm had a 2.8 increased likelihood to undergo operative treatment in the setting of calcific tendinitis of the shoulder [2].
  • Extracorporeal shock wave therapy (ESWT) can be considered as an alternative treatment for chronic calcific tendinitis of the shoulder due to its good tolerance, safety, and clinical radiological response [6].
  • Patients with calcific tendinitis of the shoulder who have factors identified for a poor outcome after ESWT should undergo a different procedure [38].
  • Utilization of barbotage as a treatment for calcific tendonitis of the shoulder appears to produce notable pain reduction in the short term (specifically at the 2-month follow-up), but begins to lose some efficacy over long-term evaluation [9].
  • The incidence of rotator cuff tears in cases of calcific tendonitis is higher than previously reported [10].
  • Routine diagnostic glenohumeral exploration does not appear beneficial in arthroscopic treatment of calcific tendinitis due to the low prevalence of intraarticular pathologies which most frequently do not require surgical treatment [8].
  • The short-term functional outcome of patients with calcific tendonitis after arthroscopic bursectomy and debridement of the calcific deposit is not influenced if performed in combination with or without a subacromial decompression [14].

Anatomy & Pathophysiology

  • Only one third of calcific tendon deposits in the general population are painful [1].
  • Calcific tendinitis of the shoulder in the Korean population has demographic, radiographic, and clinical features that are not different from those of Western populations [5].
  • Imaging and functional data indicate that calcific tendinitis of the rotator cuff with tuberosity osteolysis is a distinctive form of calcific tendinitis [11].
  • The acromion index is not different between shoulders with calcifying tendinitis, partial-thickness rotator cuff tears, or full-thickness rotator cuff tears [17].
  • The acromion index of shoulders with calcific tendinitis is comparable to that of shoulders with subacromial impingement [27].
  • The theoretical concept that a high acromion index results in increased upward force against the subacromial space, influencing pain and function in calcifying tendinitis, was not supported [40].
  • A rotator cuff tear was the only factor affecting the complete recovery of shoulder function in patients treated for calcific tendinitis [35].

Classification

  • Symptoms of calcific tendinitis can be protracted, resulting in time off work and impaired quality of life [4].
  • The demographic, radiographic, and clinical features of calcific tendinitis in the Korean population are not different from those of Western populations [5].
  • The acromion index is not different between shoulders with calcifying tendinitis, partial- or full-thickness rotator cuff tears [17].
  • Calcific tendinitis of the rotator cuff with tuberosity osteolysis is a distinctive form of calcific tendinitis [11].
  • Calcification in medial epicondylitis was more commonly identified than previously reported and was distributed over a relatively broad area [13].
  • Conservative treatment for calcific tendinitis showed clinically significant improvement, with 72% of excellent or good results regardless of the location, radiologic type and size, and initial symptoms of calcific deposits [3].
  • Shock wave therapy is a heterogeneous but effective treatment for calcifying tendinitis of the shoulder [18].

Clinical Presentation

  • Only one third of calcific tendon deposits in the shoulder are painful [1].
  • The prevalence of calcific deposits within the rotator cuff tendons is 7.8% in asymptomatic patients [21].
  • The prevalence of calcific deposits within the rotator cuff tendons is 42.5% in patients with subacromial pain syndrome [21].
  • Patients with calcific lesions >1 cm had a 2.8 increased likelihood to undergo operative treatment [2].
  • The demographic, radiographic, and clinical features of calcific tendinitis in the Korean population were not different from those of Western populations [5].
  • Acute calcific deposition of the hand and wrist is a benign self-limiting disease [15].
  • Clinical criteria predictive for shoulder rotator cuff calcific tendinopathy can help clinicians suspect this musculoskeletal disease early and with certainty [22].

Investigations

  • Calcific tendinitis symptoms can be protracted, resulting in time off work and impaired quality of life [4].
  • Shoulder surgeons should be cautious about rotator cuff tears as a comorbidity in calcific tendinitis [16].
  • Sonographic or MRI evaluation has accuracy limitations when assessing for rotator cuff tears in the context of calcific tendinitis [16].
  • The condition of articular chondrocalcinosis is characterized by multiple calcific deposits in the articular cartilage, primarily in the deeper and mid-portions of the cartilage above the tidemark [41].
  • The dynamic pathological process of calcific tendinopathy includes migration patterns of calcium deposits [12].

Treatment

  • Calcific tendon deposits are a frequent occurrence in the general population, although only one third are painful [1].
  • Nonsurgical management remains the mainstay of treatment for calcific tendinitis of the rotator cuff, with most patients improving with modalities such as oral anti-inflammatory medication, physical therapy, and corticosteroid injections [19].
  • Both operative and nonoperative treatment modalities are likely to have clinically significant improvements in function and pain, and thus it is reasonable to trial ultrasound-guided needling (barbotage) and extracorporeal shock wave therapy (ESWT) as first-line treatment [36].
  • ESWT can be considered as an alternative treatment for chronic calcific tendinitis of the shoulder due to its good tolerance, safety, and clinical radiological response [6].
  • ESWT in calcific tendinitis of the shoulder is very effective [20].
  • ESWT has a therapeutic effect on tendinosis calcarea [29].
  • Radial extracorporeal shock wave therapy (rESWT) showed a 14.28% reduction in pain, a 43% improvement in shoulder functional status, and significant increases in shoulder flexion, abduction, extension, and external rotation compared to traditional physiotherapy [26].
  • Needle aspiration of calcific deposits (NACD) is safe and effective for calcific tendinitis of the rotator cuff [30].
  • Arthroscopic debridement of calcific tendinitis with intraosseous involvement is a safe and effective treatment method similar to that of pure tendinous involvement [31].

Complications

  • Calcific tendon deposits of the shoulder are a frequent occurrence in the general population, although only one third are painful [1].
  • Shoulder surgeons should be cautious about rotator cuff tears as a comorbidity in calcific tendinitis and aware of the accuracy limitations of sonographic or MRI evaluation [16].
  • The prevalence rate of calcific deposits in the rotator cuff is 7.8% in asymptomatic patients and 42.5% in patients with subacromial pain syndrome [21].

Recovery

  • The dynamic pathological process of calcific tendinopathy includes migration patterns of calcium deposits, which are relevant to functional recovery [12].
  • Arthroscopic bursectomy and debridement of the calcific deposit provides short-term functional outcomes that are not influenced by whether subacromial decompression is performed in combination [14].
  • Outcome after arthroscopic treatment seems to correlate strongly only with the presence of residual calcium deposits in the tendon, with complete removal recommended [48].
  • Percutaneous needle aspiration and lavage is effective in the short term and in the long term in calcific tendinitis of the shoulder, with results similar to or better than those published for other techniques [23].
  • There was no significant difference in short- and long-term clinical outcomes between single- and double-needle US-guided percutaneous irrigation of calcific tendinopathy [24].
  • Utilization of barbotage produces notable pain reduction in the short term (specifically at the 2-month follow-up), but begins to lose some efficacy over long-term evaluation [9].
  • Only with one-time needling, radiologic changes in size and/or density were seen in 76.6% of the calcific tendinitis patients at 4 weeks after the index procedure [28].
  • A symptom duration of ≤10 months or calcification size of ≤10.82 mm represented the clinical scenarios most likely to show resorption after ESWT [44].
  • In ESWT studies, Type III calcifications and shorter symptom duration were associated with better outcomes [46].
  • The onset of calcium resorption in acute calcific tendinitis occurs most frequently in the summer in Japan [47].
  • The minimal and substantial clinical benefit (MCID, SCB) and responsiveness for patients with long-lasting rotator cuff calcific tendinitis treated with minimally invasive treatment options have been established for the Constant-Murley score and Disabilities of the Arm, Shoulder and Hand score [32].

Key Evidence

  • [L3] Calcific tendon deposits of the shoulder are a frequent occurrence in the general population, although only one third are painful. [1] (10.1007/s00256-015-2240-3)
  • [L3] Patients with calcific lesions >1 cm had a 2.8 increased likelihood to undergo operative treatment in the setting of calcific tendinitis of the shoulder. [2] (10.1016/j.jseint.2021.01.013)
  • [L2] Conservative treatment for calcific tendinitis of the shoulder showed clinically significant improvement, with 72% of excellent or good results regardless of the location, radiologic type and size, and initial symptoms of calcific deposits. [3] (10.1016/j.jse.2009.07.008)
  • [L3] Calcific tendinitis is a poorly understood condition in which symptoms can be protracted, resulting in time off work and impaired quality of life. [4] (10.1016/j.jse.2006.06.007)
  • [L4] This study reported demographic, radiographic, and clinical features of calcific tendinitis of the shoulder in the Korean population, which were not different from those of Western populations. [5] (10.5397/cise.2020.00010)
  • [L2] Because of its good tolerance, safety, and clinical radiological response, ESWT can be considered as an alternative treatment for chronic calcific tendinitis of the shoulder. [6] (10.1136/ard.62.3.248)
  • [L5] The primary choice of treatment for calcific tendinitis is conservative, especially in patients with acute calcific tendinitis. [7] (10.5397/cise.2020.00318)
  • [L3] Routine diagnostic glenohumeral exploration does not appear beneficial in arthroscopic treatment of calcific tendinitis due to the low prevalence of intraarticular pathologies which most frequently do not require surgical treatment. [8] (10.1186/s12891-017-1839-z)
  • [L4] Utilization of barbotage as a treatment for calcific tendonitis of the shoulder appears to produce notable pain reduction in the short term (specifically at the 2-month follow-up), but begins to lose some efficacy over long-term evaluation. [9] (10.1016/j.jseint.2024.06.005)
  • [L4] The incidence of rotator cuff tears in cases of calcific tendonitis in this cohort of patients who underwent MRI is higher than previously reported. [10] (10.1016/j.arthro.2019.11.127)
  • [L2] Imaging and functional data indicate that calcific tendinitis of the rotator cuff with tuberosity osteolysis is a distinctive form of calcific tendinitis that should be considered in clinical and surgical practice. [11] (10.1016/j.jse.2008.09.016)
  • [L5] The paper illustrates the dynamic pathological process of calcific tendinopathy, including migration patterns of calcium deposits, and discusses clinical and sonographic assessment to optimize diagnosis, rehabilitation, and interventional management for functional recovery. [12] (10.3390/diagnostics12123097)
  • [L3] Calcification in medial epicondylitis was more commonly identified than previously reported and was distributed over a relatively broad area. [13] (10.1016/j.jse.2021.08.031)
  • [L1] This study has demonstrated that the short-term functional outcome of patients with calcific tendonitis after arthroscopic bursectomy and debridement of the calcific deposit is not influenced if performed in combination with or without a subacromial decompression. [14] (10.1016/j.arthro.2015.05.015)
  • [L5] Acute calcific deposition of the hand and wrist is a benign self-limiting disease. [15] (10.1016/j.jhsa.2014.06.011)
  • [L3] Shoulder surgeons should be cautious about rotator cuff tears as a comorbidity in calcific tendinitis and aware of the accuracy limitations of sonographic or MRI evaluation. [16] (10.5397/cise.2021.00094)
  • [L3] The acromion index is not different between shoulders with calcifying tendinitis, partial- or full-thickness rotator cuff tears. [17] (10.1007/s00402-011-1263-z)
  • [L1] Shock wave therapy is a heterogeneous but effective treatment for calcifying tendinitis of the shoulder. [18] (10.1177/1941738108331197)
  • [L5] Nonsurgical management remains the mainstay of treatment for calcific tendinitis of the rotator cuff, with most patients improving with modalities such as oral anti-inflammatory medication, physical therapy, and corticosteroid injections. [19] (10.5435/jaaos-22-11-707)
  • [L1] ESWT in calcific tendinitis of the shoulder is very effective. [20] (10.1007/s00256-004-0849-8)
  • [L3] The prevalence rates of 7.8% in asymptomatic patients and 42.5% in patients with subacromial pain syndrome provide a current view on the epidemiology of calcific deposits in the rotator cuff. [21] (10.1016/j.jse.2015.02.024)
  • [L3] These clinical criteria, predictive for shoulder RCCT, can be crucial to help all clinicians suspect this musculoskeletal disease early and with certainty, thus allowing for an appropriate and prompt diagnosis path. [22] (10.1177/17585732241244515)
  • [L4] Percutaneous needle aspiration and lavage is effective in the short term and in the long term in calcific tendinitis of the shoulder, with results similar to or better than those published for other techniques, and it is only slightly invasive and painful. [23] (10.2214/ajr.07.2254)
  • [L1] There was no significant difference in short- and long-term clinical outcomes between single- and double-needle US-guided percutaneous irrigation of calcific tendinopathy. [24] (10.1148/radiol.2017162888)
  • [L2] Specifically, the rESWT group showed a 14.28% reduction in pain, a 43% improvement in shoulder functional status, and significant increases in shoulder flexion, abduction, extension, and external rotation. [26] (10.5606/archrheumatol.2019.7081)
  • [L4] The AI of shoulders with calcific tendinitis is comparable to that of shoulders with subacromial impingement. [27] (10.1007/s00167-012-2327-5)
  • [L4] Only with one-time needling, radiologic changes in size and/or density were seen in 76.6% of the calcific tendinitis patients at 4 weeks after the index procedure. [28] (10.1016/j.arthro.2013.03.033)
  • [L2] ESWT has a therapeutic effect on tendinosis calcarea. [29] (10.1007/s00776-003-0720-0)
  • [L4] Needle aspiration of calcific deposits (NACD) is safe and effective for calcific tendinitis of the rotator cuff. [30] (10.1016/j.ejrad.2016.01.018)
  • [L3] Arthroscopic debridement of calcific tendinitis with intraosseous involvement is a safe and effective treatment method similar to that of pure tendinous involvement. [31] (10.1007/s00167-022-06870-2)
  • [L2] This study established the MCID, SCB, and responsiveness for patients with long-lasting rotator cuff calci fi c tendinitis who were treated with minimally invasive treatment options. [32] (10.1016/j.jseint.2020.05.001)
  • [L3] A rotator cuff tear was the only factor affecting the complete recovery of shoulder function. [35] (10.1177/03635465231217733)
  • [L1] Both operative and nonoperative treatment modalities are likely to have clinically significant improvements in function and pain, and thus it is reasonable to trial UGN and ESWT as first-line treatment. [36] (10.1016/j.jse.2023.03.017)
  • [L3] Patients with calcific tendinitis of the shoulder who have the factors identified for a poor outcome after ESWT should undergo a different procedure. [38] (10.1302/0301-620x.99b12.bjj-2016-1178.r1)
  • [L2] The theoretical concept of a high acromion index resulting in an increased upward force against the subacromial space, which influences pain and function in calcifying tendinitis of the shoulder, was not supported. [40] (10.1007/s00167-011-1563-4)
  • [L5] The condition is characterized by multiple calcific deposits in the articular cartilage, primarily in the deeper and mid-portions of the cartilage above the tidemark. [41] (10.2106/00004623-197254040-00015)
  • [L3] A symptom duration of ≤10 months or calcification size of ≤10.82 mm represented the clinical scenarios most likely to show resorption after ESWT. [44] (10.1177/23259671241231609)
  • [L3] In SWT studies, Type III calcifications and shorter symptom duration were associated with better outcomes. [46] (10.1177/17585732251414964)
  • [L3] The onset of calcium resorption in acute calcific tendinitis occurs most frequently in the summer in Japan; however, the reasons for seasonal variation remain unclear, and further studies will be needed. [47] (10.1186/s12891-020-03773-6)
  • [L4] Outcome seems to correlate strongly only with the presence of residual calcium deposits in the tendon; complete removal is recommended. [48] (10.1016/j.jse.2004.04.001)

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